| Hypochlorhydria, or the insufficient production
of stomach acid, is a very common clinical problem with influences
far beyond the digestive tract (i.e. food and environmental sensitivity,
asthma, intestinal parasites, dermatological problems, autoimmune
disease and arthralgias.) Most practitinoers would not look at a
blood test as being their primary sources of information on the
digestive system. However, I have found it invaluable for helping
uncover my patient's digestive insufficiency.
Many factors contribute to the development of chronic hypochlorhydria
including:
- Poor food choices (Standard American Diet)
- Excess carbohydrate consumption (depletes critical
co-factors)
- Insufficient protein stimulation (veganism, low
protein diets)
- Sympathetic dominance/stress (inhibits the parasympathetic
control of digestive secretions)
- Zinc and thiamine deficiencies (essential to
the production of HCL)
- Antacid use (temporary symptom relief/worsens
problem)
- Alcohol and NSAID use (lead to gastric atrophy)
The inability to produce HCl is frequently due to a need for chloride
(low serum chloride) or a need for zinc (ALP will generally be decreased)
and thiamine (C02 will generally be decreased with an increased
anion gap). These are primary nutritional factors required for the
synthesis of hydrochloric acid. Paradoxically, sufficient HCL is
needed in order to properly absorb these nutrients as well. Hence,
the chronic nature of this problem is often perpetuated. Globulin,
albumin, and total protein will often be out of the reference range
with digestive insufficiency.
When hypochlorhydria is present, pancreatic and biliary dysfunction
are generally also present, secondary to the need for HCI. If the
food bolus is not acid enough upon exiting the stomach, it will
not trigger CCK properly and stimulate pancreatic enzyme production
and release. The subsequent improperly digested proteins and carbohydrates
contribute to the growth of opportunistic organisms and often lead
to a dysbiotic intestinal tract. Checking urinary indican levels
is an excellent means for determining protein putrefaction, carbohydrate
fermentation, and dysbiotic activity.
Hydrochloric acid supplementation is often fundamental to breaking
this cycle of chronic digestive insufficiency. Short term administration
stimulates the body’s own eventual production of HCL by reducing
the tremendous digestive stress upon an otherwise compromised gut
mucosa.
© 2004 Dicken Weatherby, ND |